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For instance, mature OLs are relatively preserved following perinatal white matter injury (e.g., periventricular leukomalacia, hypoxic-ischemic encephalopathy) in response to inflammatory and/or hypoxic-ischemic damage, as opposed to late-stage OL progenitors being highly susceptible to oxidative stress-induced apoptosis 30

Presented for research literature review only Takikawa H et al

What those studies do and do not tell us These studies are small

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The mitochondria-targeted antioxidant MitoQ ameliorated tubular injury mediated by mitophagy in diabetic kidney disease via Nrf2/PINK1

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