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mito GCL exerts neuroprotection in vivo To confirm that -glutamylcysteine exerted neuroprotection in vivo , lentiviral particles expressing wild-type or inactive (E103A) mito GCL were stereotaxically injected into the striatum of adult mice

& Zorena, K

Bauer G (2015) Increasing the endogenous NO level causes catalase inactivation and reactivation of intercellular apoptosis signaling specifically in tumor cells

This interaction reduces inhibitory binding between caveolin-1 and endothelial nitric oxide synthase, allowing increased enzyme activity and nitric oxide production in endothelial cells

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