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A critical outcome of iron-driven lipid peroxidation is the activation of ferroptosis, a regulated, non-apoptotic form of cell death characterised by the accumulation of lipid hydroperoxides (80, 81) Ferroptosis is morphologically and biochemically distinct from apoptosis, necrosis, and autophagy, and is uniquely dependent on intracellular iron availability and oxidative lipid damage (8284)
The overall TRIUMPH Phase 3 programme covers obesity, obstructive sleep apnoea, and knee osteoarthritis, and has enrolled more than 5,800 participants globally
Mechanism: VEGF upregulation drives new blood vessel formation around damaged joints
this activation increases the release of fatty acids into the blood
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