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In patients with primary or secondary hyperoxaluria, nephrolithiasis, acute or chronic oxalate nephropathy, or chronic kidney disease irrespective of aetiology, one or more of these elements are disrupted

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doi: 10.3389/fnins.2022.796129 Received 15 October 2021 Accepted 15 February 2022 Published 25 March 2022 Volume 16 - 2022 Edited by Nadja Van Camp, Commissariat lEnergie Atomique et aux Energies Alternatives, France Reviewed by Irina Dudanova, Max Planck Institute of Neurobiology (MPIN), Germany

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