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However, low concentrations of doxorubicin do not induce apoptosis in cancer cells, but rather induce cellular senescence 25,26

The review delves into the intricacies of different modification approaches, including N and C-terminal modifications, Fatty acid Side chain Modifications, and Large Molecule Conjugation Modifications, highlighting their rationale and resulting improvements in half-life, stability, receptor binding, and bioactivity

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We pretreated HepG2 cells with diamide, a well-established protein S -glutathionylation induction agent, for 2 h, followed by TNF stimulation

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