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He suspected this was due to the hormone stimulating the release of insulin independently of its ability to raise blood glucose levels

Walter have no conflicts of interest to disclose

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Advanced glycation end products (AGEs) exposure markedly impairs autophagic flux in renal TECs by activating the SMAD3/TFEB axis to suppress lysosome biogenesis and trigger lysosome depletion, leading to the accumulation of autophagosomes and SQSTM1/p62 (41)