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Once intact bacteria and other antigenic substances cross the intestinal wall, they encounter numerous immune cells that react to these substances, leading to inflammation

In preclinical studies, GIPR/GLP-1R co-agonism produces less nauseating effects compared to GLP-1R agonism alone as measured by kaolin intake in mice and rats and emetic events in musk shrews (69)

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Lack of baseline measurements for liver steatosis and fibrosis before the initiation of the EDICT study, as well as the small number of patients enrolled were the main limitations of this trial

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At the same time, levels of intracellular glutathione (GSH) deplete, reducing cells ability to defend against oxidative damage