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Amyloid- peptide is the main component of Alzheimer's plaques that can cause neuronal cell death, and it can cause oxidative damage to RBCs
The peptide's effects on growth factor signaling and vascular function may theoretically interact with certain medications affecting these pathways
Oral administration to diet-induced-obese mice for ~10 days reduces fat mass without altering food intake, decreases adipocyte size, restores insulin sensitivity, and increases adipose NAD and SAM levels [2]
In vivo, rodent studies indicate that Epithalon reduces chromosomal aberrations in aging mice, which is a marker of genomic stability [3]
Contemporary reviews consolidate evidence that 5-Amino-1MQ is a potent, selective NNMT inhibitor with broad utility as a research tool in metabolic biology [8,9]
Wang et al., 2019)
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