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This results in a lower ability to promote receptor internalization compared with endogenous GLP-1, so it allows to have an increased GLP-1R expression on the cell surface, which translates in more robust insulinotropic properties, possibly explaining the enhanced insulin secretion (in both diabetic and not diabetic human islets), induced by TZP (19) by approximately 25% more than the one induced by only one of the two agents
In chemically induced ulcer and colitis models, it supported mucosal integrity and reduced histological signs of inflammation [4]
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A recent JAMA Surgery comparison of bariatric surgery and GLP-1 receptor agonists highlights why long-term treatment selection matters
It has been studied across models of gastric ulcers, inflammatory bowel disease, intestinal fistulas, and short bowel syndrome
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