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The Kraus 2014 study reported that adipocyte-specific NNMT knockdown in mice increased adipocyte energy expenditure through a mechanism interpreted as elevated methyl-donor availability for polyamine biosynthesis and for histone methylation that modulates adipocyte gene-expression programs [2]
Dieser Genmodulationseffekt erstreckt sich auf Wege im Zusammenhang mit der Wundreaktion, Wachstumsfaktoren und antifibrotischen Mechanismen, was das Interesse in der regenerativen Biologie steigert
When you're young, the conductor is energetic and keeps everything running in perfect harmony
I., Freeman, P
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