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[7] In fibroblast and endothelial-cell contexts, FAK-paxillin signaling provides a mechanistic framework for interpreting reported changes in adhesion turnover, cytoskeletal organization, and directional movement

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BPC-157: en modelos preclnicos promueve la angiognesis regulando al alza el receptor VEGFR2, induciendo su internalizacin y activando la va VEGFR2-Akt-eNOS, con aumento de la produccin de xido ntrico (PMID 27847966)