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The effect was abolished in PepT1-knockout mice, confirming the transport pathway (PMC4957955)
It interacts with cellular pathways by facilitating copper uptake, which is essential for enzymatic activities involved in extracellular matrix maintenance
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GHK-Cu also exerts anti-inflammatory effects by inhibiting the release of pro-inflammatory cytokines (IL-6, TNF-alpha) and by modulating the activity of the nuclear factor NF-kB
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