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Patients should be instructed to report any new symptoms, unexpected effects, or concerns during treatment

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AQ undergoes rapid and extensive hepatic metabolism to its pharmacologically active metabolite DEAQ, predominantly by cytochrome P450 2C8 (CYP2C8) (Li et al., 2002)

This changes the entire release pattern

Houde AA, Lgar C, Biron S, Lescelleur O, Biertho L, Marceau S, Tchernof A, Vohl MC, Hivert MF, Bouchard L

Thus, targeting the CCL5/IL-8 signaling axis and related epigenetic-metabolic networks, such as histone lactylation and IGF2BP2, may disrupt M2 polarization cascades and restore anti-tumor immune balance