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Together, these vitamin dependent pathways maintain normal methylation and prevent excessive homocysteine accumulation
Patients with inborn errors of metabolism or reduced hepatic mitochondrial activity (mitochondrial disease) may be at an increased risk for hyperammonemia with or without encephalopathy
Source: Gojkovic et al., 2021 How BPC-157 Protects Against Alcohol Damage Four mechanisms explain why BPC-157 works across so many organ systems simultaneously
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