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GHK further inhibited TGF-1, p-Smad2, p-Smad3, and IGF-1 expression, indicating its potential as a subject of further research in pulmonary fibrosis by targeting the TGF-1/Smad 2/3 and IGF-1 pathways
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This is the consistent answer across the research peptide industry for reconstituted BPC-157
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