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Atherosclerosis progression, as a chronic inflammatory mechanism, is characterized by immune system dysregulation associated with increased pro-inflammatory cytokine production, including interleukin 6 (IL-6), tumor necrosis factor- (TNF-), and IL-1 (Vinciguerra et al., 2020

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In fact, aging as well as degenerative and neuropsychiatric diseases, such as Parkinsons disease, Alzheimers disease, autism and schizophrenia, are associated with increased ROS production, the disruption of cellular GSH pools, and downregulation of GSH-dependent enzymes (Ballatori et al., 2009

Verde, P., Casalino, L., Talotta, F., Yaniv, M

The peptide enhanced wound contraction by 11% and increased reepithelialization by 42-61% in full-thickness wound models[10]

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