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Critically, therapeutic targeting of the gut-muscle axis reduces morbidity and mortality associated with muscle dysfunction in the elderly 14 , positioning it as a key research focus for deciphering the pathophysiology of obesity-induced muscle atrophy
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To further assess their differentiation capacity, hGMSCs were subjected to osteogenic and adipogenic induction protocols [1]
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HTY has also been widely described as exerting anti-inflammatory properties primarily through modulation of signaling pathways involved in inflammation, including NF-B, mitogen-activated protein kinases (MAPKs), and redox-sensitive transcriptional regulators [7,8,9,10]
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