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KEAP1 acts as an OS sensor to regulate NRF2 turnover within the cell/body, while NRF2 activation leads to the production of antioxidant and detoxifying substances that decrease OS [66,67,68]

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From a theoretical pharmacological standpoint, combining a peptide with putative lipolytic activity (AOD 9604) with a potent triple incretin receptor agonist (retatrutide) could potentially amplify metabolic effects but this is speculative and raises the possibility of compounding adverse effects with no safety data to guide dosing or monitoring

To bridge these translational gaps, future studies should prioritize human microbiota-transplanted gnotobiotic models to recapitulate host-microbe crosstalk, coupled with longitudinal dietary interventions mimicking real-world metabolic variability