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Unfortunately, this intervention only helps about 5% of all stroke victims, even though its usefulness has been demonstrated in clinical trials [26]

There are also some data suggesting that excessive glucocorticoid action under stress can influence the endogenous GLP-1 system

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Reducing GSH biosynthesis generates replication stress in T315I-Bcr-Abl-32D cells that require dTTP/dCTP synthesis for survival, thus enabling JMF4073 susceptibility

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