Diabetic platelets show amplified platelet derived microvesicles (PMVs) release as a result of excessive oxidative stress, further enhancing coagulation and vascular inflammation
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The compound protects cells against oxidative stress and facilitates detoxification
Antimicrobial peptide LL37 and MAVS signaling drive interferon- production by epidermal keratinocytes during skin injury
Regulating HO-1 activity could be an effective strategy in treating diabetic foot ulcers, given its significance in every stage of wound healing