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Am J Reprod Immunol (1980)

Their combined effect offers comprehensive protection against toxins and infections

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H 2 O 2 induces hepatocyte premature senescence via the p53/progerin-dependent pathway To further delineate the molecular mechanism of oxidative stress-induced cell premature senescence, primary hepatocytes, isolated from normal rats, were cultured and stimulated with H 2 O 2 at the different doses (0, 10, 50, 100, 200 nM) for 24 h or at the dose (200 nM) from 0 to 24 h

Reactive oxygen species (ROS) or free radicals cause damage to key cellular components like DNA, RNA, lipids, proteins, and structural membranes