semaglutide and arrhythmia ameliorates pressure overload-induced cardiac hypertrophy by improving cardiac mitophagy to suppress the activation of NLRP3 inflammasome Contractile Effects of Semaglutide in

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The complementary nature of the desirable attributes in GLP-1, GIP and GCG led to the creation of a unimolecular hybridised triagonist with retained and balanced activity across the GLP-1R, GIP receptor (GIPR) and GCGR [1]