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These findings suggest IGF-1 LR3 promotes nitrogen retention and reduces protein breakdown, even during periods of energy restriction

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Together, the IGF-1/IGF-1R/ hsa-let-7c axis can control the odonto/osteogenic differentiation of IGF-1-treated SCAPs via the regulation of JNK and p38 MAPK signaling pathways

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Exploring the beneficial effects of GHK-Cu on an experimental model of colitis and the underlying mechanisms