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GLP-1s may not cure Alzheimers as a monotherapy, but they are reshaping how we think about the interface of neurodegenerative disease etiology, metabolic health, vascular function and they likely will cement themselves as foundational tools in delaying cognitive decline across aging populations
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Chazenbalk, G
120 Acetylation of AR at lysine 609 (K609) enhances its binding to enhancer regions of both the AR gene and the ACK1 kinase gene, even in the presence of AR antagonists
I dont necessarily like this area because there is a small chance (very small) of nerve damage (1)
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