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The Drucker review nicely reinforces that obesity biology was never single-pathway
The decrease in systolic blood pressure is aseparate entity of GLp-1 effect unrelated to weight loss [1]
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A high-salt diet contributes to kidney stone risk, at least in part, because high sodium intake raises urine calcium concentration (Sakhaee, Maalouf 2012
Xiang, S., Gu, H., Jin, L., Thorne, R
PPAR is able to upregulate the expression of almost all genes involved in the carnitine shuttle mechanism (e.g., CPT1, CPT2, CACT and CrAT) [193, 194]
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