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The identification of tubulins and protein disulfide-isomerase A3 across both cell lines suggests that X(PC) may promote cell death by targeting structural and apoptotic regulatory proteins, thereby mimicking broad caspase proteolysis activity during programmed cell death

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Astrocyte state transitions: balancing protection and pathology Traditionally, neuronal plasticity has been regarded as the principal mechanism underlying adaptive responses in the CNS, encompassing synaptic remodeling and circuit reorganization that support learning and memory