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Notably, emerging evidence suggests that excitotoxicity may synergize with immune dysregulation, exacerbating neuronal vulnerability and accelerating disease progression [99]

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However, the effect of HGF/MET on recovery from mTBI and the role of activating the HGF/MET signaling in working memory remains unclear

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Glucagon-Like Peptide-1 (GLP-1) Analog Shown to Stimulate Insulin and Suppress Glucagon Secretion in a Glucose-Dependent Manner