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Some cancer cells are very dependent on Mcl-1, which is the last line of defense keeping the cell from dying

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For instance, in NSCLC, treatment with first- and second-generation EGFR tyrosine kinase inhibitors (TKIs) often leads to the development of the EGFR T790M mutation, while third-generation inhibitors subsequently select for the C797S mutation, both of which confer resistance

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benfotiamine, a fat soluble, more physiologically active form of thiamine