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10.1016/s0140-6736(18)31788-4 Lancet
Stockwell BR, et al
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Interestingly, despite of the extracellular conformational differences induced by compound 2 and orthosteric agonists, their intracellular architectures underwent a similar reorganization of polar residues that enabled the formation TM6 sharp kink with the help of G protein binding 42,45 , indicative of GLP-1R activation (Fig
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