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Presented for research literature review only Zlatar M et al
It remains possible that nitrosyl derivatives of GSHee such as S-nitrosoglutathione and S-nitrosocysteinylglycine or S-nitrosocysteine drive an increase in fr by activating carotid body glomus cells, chemoafferent terminals in the carotid bodies, or by actions in brain sites such as the nucleus tractus solitarius 40,124 , a key nucleus of ventilatory processing
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8a, 5FU inhibits TS and can cause an imbalance in uridine-derived and deoxyuridine-derived metabolites