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The apparent contradiction between these potential protective mechanisms and clinical observations of retinopathy worsening likely reflects the complex interplay between rapid metabolic changes and pre-existing vascular damage

By contrast, l-Carnitine pretreatment could reverse PFOS-induced mitochondrial function impairment by repairing mitochondrial biogenesis, which was achieved by increasing the ATP production, maximal respiration, and spare respiratory capacity in the RTCs treated with 100-M PFOS (Fig

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Mandrekar, P., Ambade, A., Lim, A., Szabo, G