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The pathological increase in NADPH oxidase-mediated superoxide (O) and peroxide production from inflammatory phagocytic cells in the course of this inflammatory reaction acts as source of oxidative stress

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Author contributions TD designed and conducted the experiments and wrote the manuscript

7) Terminal functionalization with polyethylene glycol

Acknowledgments This work was supported by grants from the National Natural Science Foundation of China (Grant Numbers: 81572224, 81572230, 81772438, and 81671102), The National Key Research and Development Program of China, Stem Cell and Translational Research (2017YFA0105104), Science and Technology Planning Project of Guangdong Province, China (Grant Numbers: 2016A020213003, 2016B030230002, and 2017A040406007), Science and Technology Planning Project of Guangzhou, China (Grant Number: 2016201604030036), and the National Key Clinical Department, National Key Discipline, Guangdong Provincial Key Laboratory for Diagnosis and Treatment of Major Neurological Disease (Grant Numbers: 2014B030301035).

Same week, different days: - PRP (Platelet-Rich Plasma)