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However, excessive ROS cause oxidative damage to DNA, proteins, lipids, organelles, and cell membranes, which may trigger cell death pathways such as apoptosis, autophagy, ferroptosis, pyroptosis, necrosis, and ultimately lead to follicular atresia and reproductive aging [115, 116, 249, 255,256,257,258]

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Mechanism: Acts as a signal peptide that modulates fibroblast activity, collagen and glycosaminoglycan synthesis, antioxidant signaling, and copper bioavailability [1][2]

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Astrocytes as determinants of disease progression in inherited amyotrophic lateral sclerosis

Kant: Aratrma aamasnda ounlukla hayvan ve hcre verisi