Induction of autophagy-dependent ferroptosis to eliminate drug-tolerant human retinoblastoma cells
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It was among the first reports of a small molecule reproducing the metabolic effects seen with earlier NNMT-blocking approaches
Proteomics: technologies and their applications
However, in patients with aSAH, a significant imbalance arises between metabolic demand and cellular respiration, leading to an excessive accumulation of toxic free radicals that overwhelm the brains endogenous antioxidant systems, resulting in severe oxidative damage ( Following aSAH, a predominant source of ROS is the oxidative reactions involving various haem groups released during erythrolysis within the subarachnoid space
Coenzyme Q10 (CoQ10), a mitochondrial cofactor with potent antioxidant properties, has also gained attention for its role in supporting energy production and reducing oxidative damage
