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So, the exogenous application of GSH keeps the GSH/GSSG pool in check to decrease the level of membrane damage apart from increasing antioxidant machinery
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Mechanistically, HDL facilitates reverse cholesterol transport, removing excess cholesterol from arterial walls and shuttling it to the liver for excretiona process that may attenuate atherosclerosis progression [67, 68]
The Journal ofclinical investigation, 127(12), 42174227
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(Endocrinology, 2010), demonstrated a strong species-dependent effect: In rodents: High GLP-1 receptor expression in thyroid C-cells Strong cAMP signaling activation Persistent stimulation leads to tumor formation In humans and primates: Minimal or absent functional GLP-1 receptor expression in C-cells No meaningful calcitonin elevation under therapeutic exposure No observed C-cell proliferation in long-term studies Even exposure levels far exceeding clinical doses failed to reproduce rodent-like thyroid effects in primate models
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