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Lecube, A
J Neurochem (1999) 73(3):130917.10.1046/j.1471-4159.1999.0731309.x 12 PerierCTieuKGuganCCaspersenCJackson-LewisVCarelliVet alComplex I deficiency primes Bax-dependent neuronal apoptosis through mitochondrial oxidative damage
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and (4) lipid peroxidation, where free fatty acids (FFAs) overload mitochondrial -oxidation, generating cytotoxic aldehydes like 4-hydroxynonenal (4-HNE) (Mynarska et al., 2024)
These studies are internally coherent but contribute primarily supportive rather than confirmatory evidence
GLP-1 receptor agonists enhance insulin secretion in response to elevated blood glucose, suppress glucagon release, and slow gastric emptying
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