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When activated, the IRR initiates several critical cytoprotective cascades: JAK2/STAT3 and STAT5 signalling promoting anti-apoptotic gene expression and cellular survival PI3K/Akt pathway suppressing pro-apoptotic factors and promoting neuronal and endothelial cell survival NF-B modulation reducing pro-inflammatory cytokine production including TNF-, IL-1, and IL-6 MAPK/ERK activation supporting cellular repair and adaptive stress responses Anti-oxidant pathway upregulation reducing oxidative damage in metabolically stressed cells ARA-290s selective activation of the IRR without EPOR homodimer engagement allows researchers to study all of the above pathways without the erythropoietic confound present in full-length EPO studies
JOE ROGAN: Did she
NRF2, NFE2-related factor 2
They add that the pill has also proven effective in reducing the risk of major cardiovascular events such as heart attack, stroke, or death in those who are also at high risk for these events
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