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a high GSH/GSSG promotes protein deglutathionylation and a low GSH/GSSG activates Grx2 glutathionylase activity (Beer et al., 2004
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It is mediated by specific signaling molecules, notably, including receptor-interacting protein kinases 1 and 3 (RIPK1 and RIPK3) and mixed lineage kinase domain-like protein (MLKL), which drive necroptosis by activating related signaling pathways in response to severe stress or pathological stimuli (Galluzzi et al., 2018)
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