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Rett syndrome, a neurodevelopmental disorder, whole-transcriptome, and mitochondrial genome multiomics analyses identify novel variations and disease pathways
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Contrarily, CD11b upregulation in Munc13-4-knockout (KO) neutrophils was normal in response to the chemotactic peptide fMLF, a physiological stimulus that signals from plasma membrane receptors, further supporting that CD11b plasma membrane upregulation in response to CpG depends on TLR9 activation at endosomes 16
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