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It was observed that throughout the course of cerebral ischemia/reperfusion (IR), the synthesis, release, metabolism, receptors, and transmission of GABA underwent complex pathological variations (Chen et al

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8b) and grew in a similar manner to control cell lines when cultured under cystine-replete and cystine-depleted with GSH-supplemented conditions (Extended Data Fig

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Role of CYP2E1 in ethanol-induced oxidant stress, fatty liver and hepatotoxicity [online]