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Dan Stickler and Dr

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by age 60, that drops to approximately 80 ng/mLa 60% decline that directly correlates with visible aging, reduced healing capacity, and tissue degradation

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These results suggest that PS-NP exposure disrupts regulatory mechanisms within the brain-gut-microbiota axis by altering intestinal barrier integrity and microbiota composition ( in vitro studies have also demonstrated that microplastics induce more severe inflammatory responses in human microglial cells (HMC-3), leading to neuronal damage characterized by reduced cell body size and increased apoptosis ( 2.4.3 Association between microplastics and neurodegenerative disorders Microplastics can promote neuronal apoptosis and dysfunction by inducing oxidative stress and neuroinflammation ( Study ( + mice following PS-NP exposure, characterized by decreased cristae density and sparse matrix within mitochondria