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JMJD3: a critical epigenetic regulator in stem cell fate
We demonstrated that, at low doses (ng), local (intraplantar) or systemic (intraperitoneal) injections of thymulin resulted in hyperalgesia with an increase in proinflammatory mediators, and that this peptide could act directly on the afferent nerve terminals through prostaglandinE2 (PGE2)dependent mechanisms, thus forming a neuroimmune loop involving capsaicinsensitive primary afferent fibers
Once inside the cell, intracellular enzymes (thioesterases) remove the acetyl group, releasing active reduced glutathione exactly where its needed
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