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We found this paradox to be explained by GLP-1 delaying gastric emptying and the rate of carbohydrate absorption after a meal, biological actions that dominant direct effects on insulin secretion at the B-cell, both in both normal-weight individuals and in non-diabetic individuals with obesity [6, 10] (Fig
doi:10.1016/j.tem.2020.02.006 Nauck MA, Meier JJ
GLP-1 receptor signalling promotes -cell glucose metabolism via mTOR-dependent HIF-1 activation [2]
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371 In osteoblasts, the administration of GLP-1 and GIP incretins suppressed the excessive expression of the pro-degradative enzymes MMP-3 and MMP-13 induced by IL-1 stimulation
