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We used a variety of techniques, including electrophysiology, intracellular calcium imaging, dynamic cytosolic ATP measurements and assays of GLP-1 release, to investigate the mechanisms underlying the effect of glutamine on GLP-1 secretion

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These neutrophils release chemotactic agents such as oxidative leukotrienes, proteases, and platelet-activating factor, contributing to local tissue damage, fluid accumulation (pulmonary edema), surfactant inactivation, and hyaline membrane formation (Potey et al

Semaglutide and liraglutide were derived from the native structure of GLP-1

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