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doi: 10.1007/s11357-024-01449-w
In the context of MASLD and T2DM, altered metabolism is the primary etiology
When you layer reduced food intake on top of that baseline insufficiency, the gap widens quickly
ERR agonism is mechanistically distinct in that it amplifies the transcriptional machinery for oxidative metabolism rather than directly uncoupling mitochondria or stimulating adrenergic pathways
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