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Fe 2+ overload, ROS accumulation, GSH depletion, LPO, and mitochondrion shrinkage were observed in HSV-1-infected cultured human astrocytes, microglia and murine brains, which are hallmarks of ferroptosis ( HSV-1 infection enhanced the ubiquitination and degradation of Nrf2 mediated by Kelch-like ECH-related protein 1 (Keap1) so as to inhibit the expression of antioxidative genes, which disturbed cellular redox homeostasis and promoted ferroptosis (Gunderstofte et al., 2019)
Published 2022 Apr 11
If inflammation remains excessive and unmanaged, the repair environment remains chaotic
Neurotherapeutics 15(2):361370 Greco R, Demartini C, Zanaboni AM, Tumelero E, Icco R, Sances G, Tassorelli C (2023) Targeting glutamate neurotoxicity through dietary manipulation: potential treatment for migraine
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