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Oxidized PAC-quinone imine metabolites have also been shown to form GSH-conjugates which inhibit glutathione reductase (GR): the decreased activity of GR hampers the detoxification and antioxidant capacity of the GSH-GSSG cycle, further aggravating the pro-oxidative status in the cell (Ndlov et al., 2014)

The peptide downregulates NF-kB signaling, a transcription factor central to the pro-inflammatory cytokine cascade that drives degenerative change in tendons [9]

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Long-term, prospective studies are still needed, Hardaway said

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