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Role of G6PD-deficiency-mediated RBC hemolysis in ASCVD Any discussion of the potential impact of G6PD-d on ASCVD would be incomplete without considering how the predominant hemolytic phenotype may contribute to worsened disease

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The mechanism involves suppression of reactive oxygen species (ROS) that the enteric nervous system uses to trigger peristaltic contractions, combined with microbiome shifts away from butyrate-producing bacteria

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doi: 10.5694/j.1326-5377.2009.tb02371.x 200 DoorduinJde VriesEFWillemsenATde GrootJCDierckxRAKleinHC

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BPC-157 counteracted ischemic damage even when given after vessel occlusion had already occurred

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