Erythrophagocytosis by liver macrophages (Kupffer cells) promotes oxidative stress, inflammation, and fibrosis in a rabbit model of steatohepatitis: implications for the pathogenesis of human nonalcoholic steatohepatitis
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The binding affinity is so strong that the copper remains complexed with the peptide during transport and only releases at the appropriate enzymatic sites
Control samples were obtained from BioChain (San Francisco, CA, USA)
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