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AOD9604 appears to stimulate proteoglycan and collagen synthesis in chondrocytes through growth factor signaling pathways distinct from its lipolytic mechanism, supporting cartilage matrix repair in joint models
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This makes AOD 9604 useful for studying peptide-mediated regulation of adipose signaling in controlled experimental models
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Hakkola, SIRT5 is under the control of PGC-1 and AMPK and is involved in regulation of mitochondrial energy metabolism
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